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Why sunscreen is Detrimental to Black Health!



Sunscreen is detrimental to Black health! While it's true that individuals with darker skin have a natural protection against UV radiation due to higher melanin levels, sunscreen does not play crucial role in preventing skin damage for people with dark complexions unless they are using it on their palms or soles of their feet. It is a myth that sunscreen use also affects vitamin D absorption significantly and it is unnecessary for people with dark skin. White mythology wants you to believe you have no natural advantages and the petro pharmaceurical companies want the profits. In fact, sunscreen not only does not provide significant protection against UV rays it is detrimental to sea life and specifically to coral reefs!


The assertion that dark-skinned individuals need sunscreen is a misconception that has been perpetuated by a combination of white supremacist cultural beliefs, misinformation, and a lack of awareness regarding skin health. To address this topic comprehensively, we must examine several key factors: skin pigmentation, UV radiation exposure, and the role of sunscreen which is essentially black face when seen through a uv light camera.


Melanin is the pigment responsible for the color of our skin, hair, and eyes. Darker skin contains more melanin than lighter skin. Melanin provides some natural protection against ultraviolet (UV) radiation from the sun. It absorbs and dissipates UV rays, reducing the risk of sunburn and certain types of skin cancer. However, this does not mean that individuals with darker skin are completely immune to sun damage just that it is healthier for them to time their exposure to the sun rather than putting on a chemical that does more harm than good like most sun screens!


Sunscreens are formulated with various active ingredients designed to protect the skin from harmful ultraviolet (UV) radiation. However, some of these ingredients, such as oxybenzone (BP-3) and octinoxate (OMC), have raised concerns due to their potential systemic absorption and endocrine-disrupting properties. The systemic absorption of these chemicals can occur through topical application, leading to their presence in bodily fluids like blood, urine, and breast milk.


Endocrine Disruption Can Cause Gender Dysphoria and infertility


One of the primary concerns regarding certain sunscreen chemicals is their ability to act as endocrine disruptors. Endocrine disruptors are substances that can interfere with hormone systems, potentially leading to adverse health effects. Oxybenzone has been shown to exhibit estrogenic activity, which means it can mimic estrogen in the body. This mimicry can lead to hormonal imbalances that may affect reproductive health, including fertility issues in both males and females.


Studies have indicated that elevated levels of BP-3 may correlate with lower testosterone levels in adolescent boys. Although some studies found no significant associations between BP-3 exposure and fertility or reproductive hormone levels in females, the potential for disruption remains a concern due to its classification as an endocrine-disrupting chemical.


Impact on Thyroid Function

The thyroid gland plays a crucial role in regulating metabolism, growth, and development through hormone production. Research has shown mixed results regarding the impact of BP-3 on thyroid hormone levels. Some studies reported decreased levels of thyroid hormones associated with higher urinary concentrations of BP-3; Endocrine-disrupting chemicals (EDCs) are substances that can interfere with the normal functioning of the endocrine system, which regulates hormones in the body. These disruptions can occur through various mechanisms, including mimicking natural hormones, blocking hormone receptors, or altering hormone synthesis. EDCs include a wide range of chemicals such as phthalates, bisphenol A (BPA), pesticides, and polychlorinated biphenyls (PCBs).


Impact on Gender Development

Research indicates that exposure to EDCs during critical periods of development—particularly prenatal exposure—can lead to alterations in sexual differentiation and reproductive health. The endocrine system plays a crucial role in determining sex characteristics during fetal development. For instance, the presence or absence of certain hormones like testosterone is pivotal for male and female differentiation. EDCs can mimic or block these hormones, potentially leading to atypical development.


Studies have shown that exposure to EDCs can result in conditions such as genital malformations, altered anogenital distance (a marker for androgen exposure), and disrupted sexual differentiation. These changes may contribute to variations in gender identity and expression later in life, including gender dysphoria.


Gender Dysphoria and Hormonal Influence

Gender dysphoria refers to the psychological distress experienced when an individual’s gender identity does not align with their biological sex at birth. While the exact causes of gender dysphoria are complex and multifactorial—including genetic, environmental, social, and psychological factors—there is emerging evidence suggesting that hormonal influences during critical developmental windows may play a role.


Prenatal exposure to EDCs has been hypothesized to affect brain development related to gender identity. For example, animal studies have demonstrated that low doses of certain EDCs can alter brain structures associated with sexual behavior and identity. This suggests that similar effects could occur in humans exposed to these chemicals during critical periods of fetal development.


Effects on Male Reproductive Health

In addition to potential impacts on gender identity, EDCs have been linked to adverse outcomes in male reproductive health. Research has identified associations between EDC exposure and conditions such as reduced sperm quality, testicular dysgenesis syndrome (TDS), cryptorchidism (undescended testes), and increased risks for testicular cancer. These conditions may not only affect fertility but also influence perceptions of masculinity and male identity.


The concept of TDS posits that disruptions during fetal development due to hormonal imbalances caused by EDCs could lead to a spectrum of reproductive issues manifesting later in life. This highlights how environmental factors can intersect with biological processes influencing male reproductive health.


Potential Risks During Pregnancy

Pregnant women are particularly vulnerable when it comes to exposure to endocrine disruptors due to the potential impact on fetal development. Studies assessing prenatal exposure to BP-3 have generally found no significant adverse effects on offspring’s development; however, some studies reported associations with increased birth weight or other developmental outcomes. The ability of BP-3 to cross the placenta raises questions about its safety during pregnancy and its potential effects on fetal growth and development.


Chemicals in Sunscreen That Adversely Affect Pregnancy


Pregnant women need to be particularly cautious about the products they use on their skin, including sunscreen. Certain chemicals found in sunscreens have been identified as potentially harmful during pregnancy due to their ability to disrupt hormonal systems or cause other adverse effects. Here are the primary chemicals in sunscreen that should be avoided during pregnancy:


Homosalate: This chemical is known as an endocrine disruptor, which means it can interfere with the normal functioning of hormones in the body. During pregnancy, maintaining proper hormone levels is crucial for both maternal health and fetal development. Additionally, homosalate can enhance the skin’s absorption of other chemicals, increasing exposure to potentially harmful substances.


Oxybenzone: Oxybenzone is widely used in many chemical sunscreens and has been linked to several health concerns. It is absorbed into the bloodstream upon application and has been associated with low birth weights and various reproductive issues. Oxybenzone is also a photocarcinogen, meaning it can damage DNA and increase cancer risk. Studies have detected oxybenzone in breast milk, indicating that it can be passed to infants.


Octocrylene: This ingredient can also be absorbed through the skin and has been shown to produce free radicals that may lead to cellular damage. While its toxicity is considered relatively low compared to other chemicals, pregnant women may want to avoid octocrylene due to its potential effects on fetal development.


Avobenzone: Avobenzone acts as an endocrine disruptor and can remain in the bloodstream for weeks after application. Its breakdown products may cause allergic reactions and further hormonal disruptions, making it advisable for pregnant women to steer clear of this ingredient.


In summary, pregnant women should avoid sunscreens containing homosalate, oxybenzone, octocrylene, and avobenzone due to their potential adverse effects on hormonal balance and fetal health.

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